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In nerves exposed to a hundred Hz when bathed in 10 mM glucose, the CAP failed suggesting that even within the presence of normoglycemic glucose additional vitality substrate is required when the tissue vitality demand will increase. Such information are important as they present that glycogen has an important position below physiological conditions. The truth that glycogen supported CAP conduction within the absence of exogenously utilized glucose in all probability has no value in ascribing a task to glycogen, since aglycaemia is an artificial experimental procedure, and hypoglycemia is an iatrogenic consequence of insulin therapy that didn't exist previous to 1922 (Frier et al., 2014), thus cannot be glycogen’s principal role. However supplying lactate to axons when tissue energy demand increases implies a novel position for brain glycogen and suggests it acts as an energy buffer relatively than an vitality reserve, whose function is not to single-handedly assist conduction, however to act in live performance with ambient ranges of normoglycemic glucose.

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